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3.
Journal of Korean Medical Science ; : 708-711, 2000.
Article in English | WPRIM | ID: wpr-171764

ABSTRACT

A patient with a fulminant amebic colitis coexisting with intestinal tuberculosis had a sudden onset of crampy abdominal pain, mucoid diarrhea, anorexia, fever and vomiting with signs of positive peritoneal irritation. Fulminant amebic colitis occurring together with intestinal tuberculosis is an uncommon event and may present an interesting patho-etiological relationship. The diagnosis was proven by histopathologic examination of resected specimen. Subtotal colectomy including segmental resection of ileum, about 80 cm in length, followed by exteriorization of both ends, was performed in an emergency basis. Despite all measures, the patient died on the sixth postoperative day. The exact relationship of fulminant amebic colitis and intestinal tuberculosis is speculative but the possibility of a cause and effect relationship exists. Fulminant amebic colitis may readily be confused with other types of inflammatory bowel disease, such as idiopathic ulcerative colitis, Crohn's disease, perforated diverticulitis and appendicitis with perforation. This report draws attention to the resurgence of tuberculosis and amebiasis in Korea, and the need for the high degree of caution required to detect it.


Subject(s)
Humans , Male , Journal Article , Diagnosis, Differential , Dysentery, Amebic/surgery , Dysentery, Amebic/pathology , Dysentery, Amebic/diagnosis , Dysentery, Amebic/complications , Fatal Outcome , Middle Aged , Tuberculosis, Gastrointestinal/surgery , Tuberculosis, Gastrointestinal/pathology , Tuberculosis, Gastrointestinal/complications
5.
GEN ; 46(1): 34-9, ene.-mar.1992. ilus, tab
Article in Spanish | LILACS | ID: lil-111114

ABSTRACT

Se describen 5 casos de Colitis Amibiana Necrotizante, ocurridos en un período de tres años en el Hospital Universitario de Caracas, en todos los servicios de Cirugía. En tres de dichos casos se planteó el diagnóstico durante el pre-operatorio, iniciándose tratamiento anti-amibiano, sobreviviendo sólo uno de ellos. Se revisan las manifestaciones clínicas métodos diagnóstico y tratamiento de la enfermedad


Subject(s)
Adult , Middle Aged , Humans , Male , Female , Dysentery, Amebic/pathology , Dysentery, Amebic/therapy , Necrosis
6.
GEN ; 45(1): 65-8, ene.-mar. 1991. ilus
Article in Spanish | LILACS | ID: lil-113340

ABSTRACT

El ameboma es una complicación rara de la ambiasis intestinal que requiere de su sospecha y diagnóstico preciso para instalar el adecuado tratamiento médico específico, que permita al paciente una mejor evolución


Subject(s)
Humans , Male , Adult , Middle Aged , Dysentery, Amebic/diagnosis , Diagnosis, Differential , Dysentery, Amebic/parasitology , Dysentery, Amebic/pathology , Dysentery, Amebic/therapy
7.
GEN ; 44(2): 155-8, abr.-jun. 1990. ilus
Article in Spanish | LILACS | ID: lil-107974

ABSTRACT

La colitis amibiana fulminante es una complicación infrecuente de la amibiasis la cual está asociada a una alta tasa de mortalidad. La existencia deuna masa tumora (ameboma) en recto o colon es de hecho muy rara pero cuando se presenta puede causar dificultades diagnósticas. Reportamos el caso en una paciente embarazada de 27 años de edad, quien fue intervenida de emergencia presentando esta rara aosciación


Subject(s)
Amebiasis/pathology , Dysentery, Amebic/pathology , Pregnancy Complications
8.
Article in English | IMSEAR | ID: sea-24641

ABSTRACT

Histopathological changes in intestinal and hepatic amoebiasis were studied in Swiss albine mice with and without silica pretreatment. The intestinal infection rate was higher in silica treated mice (83%) as compared to non-silica treated mice (33%). Histologically all the silica treated mice had caecal ulceration which tended to spread across the muscularis propria into the serosal layer. The hepatic lesions were observed grossly in 100 and 60 per cent mice with and without silica treatment, respectively. However, the liver lesions in the silica treated mice were more severe, leading to satellite abscesses and in some cases the capsule ruptured leading to adhesions to the diaphragm and peritoneum. Histologically, the blockade of histiocytes with silica resulted in mainly a neutrophilic damage which was extensive and widespread. It also destroyed the amoeba and led to abundant fibrosis. In the non-silica group the damage was slow and devoid of neutrophils with quicker healing and lack of fibrosis resembling human hepatic lesions.


Subject(s)
Animals , Cecum/pathology , Disease Models, Animal , Dysentery, Amebic/pathology , Liver/pathology , Liver Abscess, Amebic/pathology , Mice , Silicon Dioxide/pharmacology
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